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CDCA: chenodeoxycholic acid

Oxidative stress, in turn, induces cellular impairment and apoptosis by means of bioenergetic failure (via mitochondrial damage), depletion of intracellular antioxidants, biomolecular damage, microtubular damage, activation of ion channels, demyelination, neuroinflammation, and impaired mitophagy [10,11,12,13,14]

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Oxidative and endoplasmic reticulum stress responses to chronic high-altitude exposure during the development of high-altitude pulmonary hypertension

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