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Prolonged alterations in iron metabolism can lead to organ dysfunction, particularly affecting the liver and potentially resulting in heart failure [28]

We then move to repair by targeting mitochondrial dysfunction, chronic neuroinflammation, glymphatic stasis, and synaptic loss through multimodal bio-interventionsspecifically advanced Photobiomodulation, ketogenic metabolic switching, electromagnetic field therapies, and targeted peptide supplementation

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TUNEL-positive cells and 4-HNE accumulation were observed within TLS in both sulfasalazine-treated and vehicle-treated mice, while cleaved caspase-3, an apoptosis marker, was not evident in either group (Figure 5C)

J Radioanal Nucl Chem 249(1):3337