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In fact, SOX9 was destabilized by the downregulation of selenium-responsive PRMT5 that sustains SOX9 stability via methylation 93
Similarly, in a model of neurotoxicity, excess GPx-1, as well as antioxidants, such as MitoQ, attenuated cellular ROS, thereby leading to a decrease in activation of the caspase cascade and a reduction in apoptotic cell death (191)
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John Salerno, who specializes in using an integrative and functional approach to address patient health concerns
10.1111/obr.13053 [DOI] [PMC free article] [PubMed] [Google Scholar] 64.Ahmad SA, Al Thobiti TA, El Toum M, Al Harbi F
