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Endoplasmic reticulum stress and oxidative stress in cell fate decision and human disease

NNMT influences this regulatory landscape via: Reducing SAM availability for DNA and histone methylation Causing altered chromatin states that can activate oncogenes or silence tumor suppressors Facilitating oncogenic reprogramming in both tumor cells and stromal microenvironment 9

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Akathisia among patients undergoing antipsychotic therapy: prevalence, associated factors, and psychiatric impact

We discovered that RidA functions as a highly efficient and ATP-independent chaperone holdase under HOCl-stress conditions [15]