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[DOI] [PMC free article] [PubMed] [Google Scholar] 208.Zhang Z., An R., Guo H., Yang X
Hyperglycemia-induced epigenetic reprogramming in pulmonary DCs suppresses MHC-II and costimulatory molecule expression via histone hyperacetylation, blunting antigen presentation and CD8 + T-cell priming (Figure 5)
Recently, specific inhibitors of ACSL4 and research on its upstream transcriptional regulators such as SP1 have shown clear cartilage-protective effects in OA models, suggesting that future strategies combining GPX4 modulation to enhance defense with ACSL4 inhibition to reduce attack may achieve more potent and synergistic therapeutic outcomes (He W
Expression of p11 in patients with depression
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