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Incretin based treatments and mortality in patients with type 2 diabetes: systematic review and meta-analysis

Moreover, we excluded patients and controls who suffered from neurodegenerative and neuroinflammatory disorders, such as chronic fatigue syndrome [54], Parkinsons or Alzheimers disease, multiple sclerosis, stroke, or systemic (auto)immune diseases such as diabetes mellitus, psoriasis, rheumatoid arthritis, inflammatory bowel disease and scleroderma, and liver and renal disease

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These data suggest that high glucose levels promoted the proliferation of activated hPSCs through the TGF signalling pathway, and GSH has the potential to attenuate the activation phenotype of hPSCs

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Similar content being viewed by others Background Nonalcoholic fatty liver disease (NAFLD) is an important cause of chronic liver injury worldwide [1, 2]