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Upon IB degradation, NF-B is released and translocates into the nucleus, where it recruits p300, which in complex with cAMP response element-binding protein acetylates RelA/p65, thereby activating NF-B transcription and enhancing the expression of downstream pro-inflammatory genes (Lawrence 2009)

PGC-1, as a key regulator of mitochondrial biogenesis and transcriptional regulator, is involved in the regulation of BACE1 transcription

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