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Reactive astrogliosis in neuroinfection is thus defensive and neuroprotective: inhibition of astrocytic reactivity, for example by knocking out GFAP, 231 or by genetic deletion of NK-1R receptor for substance P, which suppresses reactive astrogliosis that defines resistance to N

In general (although of course there are nuances), a high ratio of reduced glutathione (GSH) to oxidised glutathione (GSSG) in the blood indicates high redox (antioxidant) capacity/reducing power

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APOE2, E3 and E4 differentially modulate cellular homeostasis, cholesterol metabolism and inflammatory response in isogenic iPSC-derived astrocytes

In some cases, a systemic approach may be taken depending on your overall goals