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Reactive protein isoforms differ in their effects on thrombus growth

Abstract Helicobacter pylori infection is a major etiological factor in gastric diseases

GSH augmentation as a potential therapeutic strategy in CNS disease has been suggested for decades, 23 although repletion efforts have been hindered by inability to assess human CNS GSH concentrations in vivo and poor oral absorption of GSH

On the other hand, astrocyte depletion in the chronic phase of MS resulted in disease attenuation and reduced inflammatory infiltrate (107)

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