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The possible mechanism underlying the increased oxidative stress might be mediated by complex I which is not only overexpressed in FTD3 neurons but also produces large amounts of superoxide when the matrix NADH/NAD + ratio is elevated as a result of increased TCA cycle activity (as also seen in diseased neurons)
J Biol Chem 281:1506415072 Cline GW, Lepine RL, Papas KK, Kibbey RG, Shulman GI (2004) 13C NMR isotopomer analysis of anaplerotic pathways in INS-1 cells
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Dainichi T, Kitoh A, Otsuka A, Nakajima S, Nomura T, Kaplan DH, Kabashima K
6, Sept
