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80 It has been reported that impaired thioredoxin expression in spontaneously hypertensive rats 81 and in endothelial cells negatively regulates ROS production in VSMC through thioredoxin upregulation

Moreover, hesperetin and hesperidin are reported to directly suppress viral replication in other pathogens such as RSV, zika virus (ZIKV), chikungunya virus (CHIKV), suggesting them as potential broad-spectrum antiviral agents with multi-target mechanisms (302, 303)

They contribute to the release of pro-inflammatory mediators, cell activation, and phagocytosis, and are involved in sensing and amplifying the inflammatory signal

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Additionally, this approach activated the protein kinase R-like endoplasmic reticulum kinase (PERK)/active transcription factor 4 (ATF4) signaling pathway, crucial for endoplasmic reticulum stress regulation, and partially mitigated hepatic lipid accumulation

It is important to note that while these genetic technologies can identify how genetic variations relate to drug responses, drug responses are influenced by a bunch of other factors, such as protein expression, how the body metabolizes drugs, and epigenetic regulation