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It is possible that dysregulated glutathione metabolism in mitochondria may promote necroptosis by altering the function of PGAM5 (phosphoglycerate mutase family member 5) and DRP1 (dynamin-related protein 1), mitochondrial proteins that are known to be associated with the necrosome (21, 22)

In turn, these perturbations mediate changes in transcriptome and proteome profiles, as well as morphological changes, for instance in spine remodeling [15]

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