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In the current study, we conducted a quantitative proteomic analysis to explore the pathways mediating the therapeutic effect of GSH in cerebral ischemia/reperfusion (I/R) model rats

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Methods The study cohort included 110 patients with acute relapse of schizophrenia and 55 healthy controls

Aims/hypothesis Barth syndrome is a mitochondrial disorder caused by Tafazzin (TAZ) mutations, which impair cardiolipin remodelling and contribute to systemic metabolic alterations

Specifications: Purity: 99% Appearance: White lyophilized powder Solubility: Fully soluble in sterile water (HO) Quality Verification: RP-HPLC validated Storage: 28C (refrigerated after reconstitut