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Previous studies have elucidated that the TGF-/SMAD pathway mediates apoptosis of vascular endothelial cells ( TGF- may also induce CNI-related ED via non-SMAD signaling pathways, such as the RhoA/ROCK, RAS/MEK/ERK, and PI3K pathways ( via catalyzing the deacetylation of acetyl-L-lysine side chains in histones ( via cooperating with TGF- after CNI, which also supports the above hypothesis
The patient's presentation is classic for hypokalemic periodic paralysis , an autosomal dominant disorder most frequently caused by mutations in skeletal muscle voltage-gated calcium channels
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Puerarin prevents high glucose-induced apoptosis of Schwann cells by inhibiting oxidative stress
A necessary striking synergistic effect may not be achieved
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