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Plasma levels of TMAO can be increased with healthy and unhealthy diets and do not correlate with the extent of atherosclerosis but with plaque instability

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In addition, intestinal macrophages respond to ACh released by nerve fibers in the intestinal myenteric plexus, ameliorating inflammation in vivo , in a mechanism that involves the activation of Jak2/STAT3 and the transactivation of STAT3-responsive DNA elements (84)

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Macrophages exposed to choline deficiency or ChoK inhibitors (MN58b or RMS932A) exhibit poor PC and sphingomyelin mitochondrial membrane composition, leading to defective mitochondrial ATP synthesis that boosts AMPK activation and mitophagy ( Figure 3 )

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