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The change from baseline for GSH+GSSG Protein (p = 0.034) and GSH+GSSG Total (p = 0.006) were greater following NLNG than the placebo

Mitochondrial translation suppression enhanced immune response against pathogen infection, and we show that C32E8.9 is responsible for this immune regulation by mediating sma-4 , which is essential for supporting lifespan extension upon mitochondrial translation inhibition

gossypii to form riboflavin and ArP, which is recycled in the riboflavin biosynthetic pathway (Figure 1)

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