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Although many of the VacA-mediated effects either directly or indirectly result from membrane binding and pore formation, VacA also enters the cytosol, subsequently accumulates in the mitochondrial inner membrane, and activates endogenous mitochondrial channels, thereby inducing apoptosis (105, 212, 329, 691)

Sunakawa H., Mizoi K., Takahashi R., Takahashi S., and Ogihara T., Impact of PGlycoproteinmediated Drugendogenous Substrate Interactions on Androgen and Bloodbrain Barrier Permeability, Journal of Pharmaceutical Sciences 113, no

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This affects mRNA stability and translation efficiency