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On the other hand, Epitalon's core proposed mechanism activating telomerase is the same pathway cancer cells exploit to achieve unlimited replication, since telomerase reactivation (often via TERT promoter mutation) is one of the defining features that allows tumor cells to bypass normal senescence limits

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In these host cells, as mentioned before, the pathogen produces antibiotic persisters that survive exposure to the fluoroquinolone enrofloxacin better ( Salmonella

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