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These findings suggested that P-Mn/TI@mHSA modulated plaque macrophage activity, alleviated the inflammatory response, and subsequently inhibited the recruitment of additional immune cells, such as monocytes, to the plaque

Importantly, DNase I digestion, which degrades exosomal dsDNA, markedly suppressed STINGTBK1IRF3 signaling, confirming the functional contribution of exosomal dsDNA in HSC activation (Supplementary Fig

Effects of neurotransmitters, gut hormones, and inflammatory mediators on mucus discharge in rat colon

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