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118,144,145,146,147 These findings suggest that A deposition or plaque formation might not be the actual cause of the disease, but rather a result or secondary factor of the pathological process

Chemicals such as ATP, TNF- , prostaglandin E2, NGF, bradykinin, serotonin and protons are released by mast cells, macrophages, and epithelial cells to induce peripheral sensitization, which serves to increase pain awareness to limit further damage, fight infection and initiate injury repair

[DOI] [PMC free article] [PubMed] [Google Scholar] Schoenfeld S

CD44v contain additional peptide motifs that can interact with and sequester growth factors and cytokines at the cell surface thereby functioning as coreceptors to facilitate cell signaling

[DOI] [PubMed] [Google Scholar] 111.Mixed lineage kinase inhibitor CEP-1347 fails to delay disability in early Parkinson disease

LSECtin on tumor-associated macrophages enhances breast cancer stemness via interaction with its receptor BTN3A3