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In some cells, ferroptosis acts as an active driver of tissue destruction, whereas in others, it represents a passive consequence or an accompanying phenomenon of the pathological environment
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Knockdown of PAX-interacting protein 1 ( PAXIP1 ) or nucleosome remodeling factor CHD4 restored cisplatin resistance in BRCA2 -deficient B lymphocytes and an ovarian cancer cell line via inhibition of recruitment of MRE11A and diminished degradation of replication forks, independent of HR [27, 28]
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