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A subsequent study revealed that paclitaxel increased the number of Na v 1.7 channels at the nerve endings by enhancing the anterograde transport of Na v 1.7 vesicles to distal axons (Akin et al., 2021)

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This pathway consumes a substantial share of labile methyl groups

doi:10.1016/S0140-6736(20)32667-2

Furthermore, the NADPH-producing metabolic enzyme ME1 which links glycolysis with the TCA cycle was reported to promote the metastatic potential of HCC and correlated with poor prognosis [102]