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The progression of atherosclerotic plaque is defined by three key features: the formation of a fibrous cap that covers the lesion under the endothelial cell layer, development of a necrotic core, and calcification

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10.1111/hepr.13433 69 LiuR.LiY.TianL.ShiH.WangJ.LiangY.et al (2019)

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Its epigenetic modulation capabilities also suggest it can influence DNA methylation patterns and histone modifications, potentially providing long-term regulatory mechanisms for inflammatory processes [44]