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This underscores the therapeutic potential of enhancing the functionality of the antioxidant axis to treat UC targetting ferroptosis ( Nrf2 /GPX4 signaling, increasing GPX4 activity, inhibiting ferroptosis, and improving intestinal barrier integrity ( Nrf2 /GPX4 signaling, increase GPX4 activity, inhibit ferroptosis, and treat UC ( In summary, drugs can upregulate the content of GSH and GPX4 through various signaling pathways, thereby treating UC by inhibiting ferroptosis and promoting antioxidation

Heyland D, Muscedere J, Wischmeyer PE, Cook D, Jones G, Albert M, Elke G, Berger MM, Day AG, Canadian Critical Care Trials Group

The Recovery, Interpretation and Use of Ancient Pathogen Genomes. Current Biology 30, no

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