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440 This combination selectively targets cancer cells at lower doses, offering an effective, less toxic strategy against apoptosis-resistant cancers

Phosphorylation site mutations in the human multidrug transporter modulate its drug-stimulated ATPase activity

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In the SOD2 C/T (Thr 58 Ile) polymorphism, the 58 Ile amino acid has been indicated as favourable in comparison with the 58 Thr form, reducing the vitiligo risk (Laddha et al

F344 rats treated with pipermethystine (10 mg/kg) for 2 weeks resulted in significant increase in hepatic glutathione, cytosolic superoxide dismutase (Cu/ZnSOD), tumor necrosis factor alpha mRNA expression, and cytochrome P450 (CYP) 2E1 and 1A2, suggesting that pipermethystine caused adaptation to oxidative stress and possible drug-drug interactions (10)

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