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Description

Therefore, LLT may be appropriate for all DHRs correlated with T cells, regardless of the background mechanism

Patient population stratification: which patients will benefit most (frail older, endocrine decline, metabolic syndrome)

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Implications Resistance mechanisms that undermine the immunomodulatory effects of CD38targeting therapies can be tumor intrinsic, such as the downregulation of CD38 surface expression and expression of complement inhibitor proteins, and immune microenvironmentrelated, such as changes to the natural killer (NK) cell numbers and function in the bone marrow niche