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Obesity-related adiponectin deficiency contributes to mitochondrial dysfunction and a low-grade inflammatory state which has been implicated in COPD, asthma, bacterial and viral pneumonia, and aspergillosis [89]

These findings highlight a critical role for ferroptosis in promoting COVID-19 lung pathology and suggest that suppressing this iron-dependent cell death may be beneficial

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As mentioned above, P53 can enhance the susceptibility of cell to ferroptosis by inhibiting SLC7A11 and regulating cystine metabolism and GSH synthesis, revealing P53s new role in tumor suppression and offering a basis for developing antitumor strategies targeting ferroptosis.Given P53s key role in regulating SLC7A11 and ferroptosis, restoring its function is a promising therapeutic approach.Adeno - associated viral vectors can reintroduce wild - type P53 into glioma cells to reverse abnormal SLC7A11 expression and increase ferroptosis sensitivity.Additionally, combining P53 activators with SLC7A11 inhibitors may boost therapeutic efficacy