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This reaction simultaneously depletes two critically important metabolic cofactors: SAM (the universal methyl donor for hundreds of epigenetic and biosynthetic reactions) and nicotinamide (the precursor for NAD+ biosynthesis via the salvage pathway)

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N-acetylcysteine reverses the mitochondrial dysfunction induced by very long-chain fatty acids in murine oligodendrocyte model of adrenoleukodystrophy