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By obstructing the phosphorylation of nearby effector molecules like ZAP70, these phosphatases combat TCR signaling, leading to the eventual exhaustion of T cells [103]

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364 The affected astrocytes demonstrate profound loss of function, in particular impaired K + buffering, 365,366 compromised H + transport, 367 and substantial decrease in glutamate uptake

Harding, G., Coyne, K

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