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Description
In the tumor microenvironment, chronic inflammatory factors (e.g., TNF-, IL-1) or ROS activate NF-B, which binds to the promoter region of the Lon gene to enhance its transcription, forming a NF-B Lon upregulation positive feedback loop
For instance, NF-B, a crucial mediator of inflammation, induces transcription of pro-inflammatory factors, leading to synaptic dysfunction and neuronal loss
A.BolonB
doi: 10.1016/j.foodres.2021.110602
Aberrant methylation of an imprinted tumor suppressor gene ARHI in human breast cancer cells
CCk-8 test results showed that US-HA-Exo could mitigate A toxicity to neurons in vitro
