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Hu S, Rayman MP

The required duration of the HL pre-treatment was remarkably short since a portion of the R-sustained genes was upregulated in R only after two min of HL pretreatment (Figure 5A)

By inhibiting CD38 activity in astrocytes genetically or pharmacologically, NAD + levels were increased, transcriptional reprogramming was inhibited, and monocyte chemotactic activity was impaired [49]

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Mesencephalic astrocyte-derived neurotrophic factor (MANF) protects against A toxicity via attenuating A-induced Endoplasmic reticulum stress