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Results Despite impaired cardiolipin remodelling, pancreatic islets from Taz-KD mice maintained insulin secretion, supported by compensatory mechanisms such as increased glucose uptake, expanded mitochondrial volume and increased metabolic parameters

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From its origins in structure-guided drug design to its current applications across multiple research disciplines, 5-amino-1MQ exemplifies how targeted enzyme inhibition can provide insights into complex biological systems while maintaining the specificity required for rigorous scientific investigation

also aids in natural cell detoxification by protecting cells from free radical damage