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Molecular remedy of complex I defects: rotenone-insensitive internal NADH-quinone oxidoreductase of Saccharomyces cerevisiae mitochondria restores the NADH oxidase activity of complex I-deficient mammalian cells

This section and Table 4 include different potential targets specific to different ROS-mediated cellular processes or pathways, as well as potential therapeutic strategies for mitigating cancer

The volume of the dialysis buffer should be a minimum of 20 times the sample volume

All three drugs aim to enhance or mimic dopamine activity in the brain

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