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doi: 10.1146/annurev-pharmtox-010818-021524 [DOI] [PubMed] [Google Scholar] 79.Bates D, Schultheis BC, Hanes MC, Jolly SM, Chakravarthy KV, Deer TR, et al

One simple explanation of the link between decreased GSH:GSSG and enhanced glutathione synthesis is that increases in the chloroplast glutathione redox potential allow GRX-mediated oxidative activation of both APR and -ECS (Figure 3A)

Further, susceptibility to ferroptosis appears to be connected to the differentiation of melanoma cells and cannot necessarily be applied to metastatic-derived cells

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853 Activating the Nrf2 pathway to negatively regulates ferroptosis may also be a possible future strategy for treating lung injury caused by intestinal I/R injury