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We then treated HepG2 cells with the lysosomal inhibitor Chloroquine (CQ) or the proteasome inhibitor MG132 and tested whether TFAP4 is also regulated by lysosome- or proteasome-mediated protein degradation in response to nutrient availability

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Several mechanisms likely play a role in the pathogenesis of lichen sclerosuss inflammatory hypopigmentation: (1) decreased melanin production, (2) block in transfer of melanosomes to keratinocytes, and (3) melanocyte loss [56]