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Once-weekly semaglutide in adolescents with obesity

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AOD-9604 and the beta3-adrenergic receptor pathway Chronic AOD-9604 administration in obese rodent models, specifically 2 mg/kg body weight daily via intraperitoneal injection for 14 days, upregulates beta3-AR RNA expression in white and brown adipose tissue to levels observed in lean mice

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Proposed mechanisms of MOTS-c: Activates AMPK Improves insulin sensitivity Enhances glucose utilization Increases metabolic flexibility Improves mitochondrial efficiency Supports exercise performance Why AMPK matters: AMPK is often called the body's metabolic master switch