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An alternative toxin-based model of demyelination is the dietary consumption of cuprizone that causes demyelination of specific brain white matter regions, such as corpus callosum and hippocampus [20]

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This is the first time the active substance is metabolised in the liver, which is called the first pass metabolism

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Systematic reviews of BPC-157 in musculoskeletal applications identified 36 studies demonstrating improved functional, structural, and biomechanical outcomes across muscle, tendon, ligament, and bone injuries in preclinical models