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Description
Natural polysaccharides can also delay aging by reducing oxidative damage and inflammatory effects and inhibiting telomere shortening in different aging models (Table 2)
In addition to this attack, which might be caused by homocysteinylation of the vascular wall proteins by homocysteine and/or its oxidized products (homocysteic acid, homocysteine sulfinic acid, and homocysteine thiolactone), in vitro and in vivo work suggests that reduced homocysteine can inhibit endothelial cell turn-over (Wang et al, 1997) and promote smooth muscle cell proliferation (Lubec et al, 1996b
2010;299(1):H193201
Postnatal microbial colonization programs the hypothalamic-pituitary-adrenal system for stress response in mice
These findings were supported by the relaxation of large vessels (ex vivo), mainly by acting on vascular endothelial cells but also on vascular smooth muscle cells, increasing NO production and migration of endothelial cells, activating the signal pathways of Src, Cav-1, and eNOS, and reducing proteinprotein interaction between eNOS and Cav-1 [121,122]
Colon Rectum 57 , 10251048 (2014)
