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Human glioblastoma cells responded to inhibition of fatty acid oxidation with increased ROS, ATP depletion, and cell death (Pike et al

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The results are consistent with our understanding of H 2 in ERW

It can neutralize free radicals, support immune function, and detoxify harmful substances

Tumor metabolism has been considered a downstream manifestation of oncogenic pathways, but increasing evidence supports metabolic dysregulation as a driving force in tumor initiation and progression

All these approaches are reported to have a less-negative impact on EF, with many of the studies reporting a complete recovery at one-year follow-up [318]