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Anti-CD3 scFvs isolated from additional selection and screening can be benchmarked against this current standard

Braakhuis et al

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Walker EF, Brennan PA, Esterberg M, Brasfield J, Pearce B, Compton MT

3.4 Connections between genetic mutations and metabolic changes The HPGL/PCC syndrome, where loss-of-function mutations affect SDH and increase intracellular levels of the oncometabolite succinate, thus causing onset of pheochromocytoma and paraganglioma, is a proof-of-concept that metabolic changes can drive tumorigenesis

The next frontier lies in decoding the spatiotemporal logic of hepatic mitochondrial signaling, specifically how transient metabolic stress and chronic injury are transduced into distinct systemic messages through mitokines, metabolites, and ox-mtDNA