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Description
ChaC1, which is referred to as Botch in the report [113], is induced and promotes neurogenesis by antagonizing Notch signaling in mouse models

Riboflavin is critical to the proper functioning of mitochondrial pathways, and mitochondrial dysfunction linked to riboflavin deficiency is linked to many neurological conditions, including AD, PD, MS, Alper's syndrome, and Kearns Sayre Syndrome, reviewed in Ref

Carbamoylation correlates of cyanate neuropathy and cyanide poisoning: relevance to the biomarkers of cassava cyanogenesis and motor system toxicity

Front Immunol 12

doi: 10.1055/s-2005-837780

The structure was based on phase and amplitude Fourier component information from cryo electron microscopy (cryoEM) images and electron diffraction, respectively
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