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Coupled with genetic and epigenetic profiling, these data may help clinicians identify individuals at higher risk for oxidative damage-related cognitive decline, enabling earlier and more tailored nutritional and lifestyle recommendations

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Biofactors 30, 159169

By interfering with the anaplerotic use of glutamine in the Krebs cycle, pharmacological inhibition of Glu dehydrogenase by epigallocatechin gallate and R162 or amino acid transaminase by aminooxyacetate slows tumor growth and may be helpful in vascular hyperproliferative disorders [313]

It is important to point out the interaction between MTHFR polymorphisms and environmental risks for MDD, such as dietary and stress

For instance, Golgi -mannosidase II (GMII