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doi: 10.1016/j.cell.2016.06.001

However, the hydroxyl group can affect the turnover of non-glutathione disulfide substrates

It was also reported in DRG neurons of wild type and TRPV4 knockout mice that activation of TRPV4 by paclitaxel induced mechanical allodynia and excessive ROS production, although the allodynia and oxidative stress was partially decreased by the TRPV4 antagonist treatment (Materazzi et al., 2012) (Table 1)

These changes disrupted mitochondrial dynamics, leading to structural abnormalities, such as disorganized cristae, and excessive ROS production [187]

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