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Mechanistically, SLC25A39 deficiency induced mitochondrial dysfunction, characterized by excessive mitochondrial reactive oxygen species (ROS), reduced membrane potential, diminished adenosine triphosphate (ATP) synthesis, aberrant mitochondrial permeability transition pore (mPTP) opening, and cytochrome c release

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Key benefits are: Sebum Regulation: Facilitates the uptake of fatty acids into mitochondria, where they are converted into energy rather than sebum, effectively reducing excess sebum production

See INGREDIENTS section for full list of ingredients