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At this juncture, one might consider whether the glucose catabolic pathway harbors exploitable vulnerabilities
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The same authors also showed [59, 70] that the action of both CAT and GPx was insufficient to explain the erythrocyte oxidative catabolism, and proposed [70] a model including Prx2 that, in accordance with their experimental data, could better explain the erythrocyte antioxidant defense system
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