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Therefore, enhanced glutaminolysis might eventually contribute to the initiation and progression of cancer by stimulating cell growth via the mTORC1 pathway and enhancing aerobic glycolysis while disrupting the proper elimination of misfolded proteins, damaged DNA and organelles through the inhibition of autophagy and the DNA damage response 166

Zhu et al., 1999a)

Exclusion criteria included non-relevance, poor sample size, and inconclusive data and significance

Table 1 summarizes key recent studies on eosinophil involvement in IBD across the discussed categories, highlighting their findings and clinical implications

Mol Cancer 17(1):111, 2018

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