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Conversely, excessively high intracellular K + in tumor cells may inhibit the antitumor abilities of tumor-associated macrophages, a process possibly achieved through the readjustment of OXPHOS and glycolysis

DNA repair mechanisms and their clinical impact in glioblastoma
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[DOI] [PMC free article] [PubMed] [Google Scholar] 56.Newport M.T., VanItallie T.B., Kashiwaya Y., King M.T., Veech R.L
