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Activating AhR alleviates cognitive deficits of Alzheimer's disease model mice by upregulating endogenous A catabolic enzyme Neprilysin

Both have similar side effect profiles, most commonly nausea and digestive discomfort, especially during dose escalation

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The resulting imbalance affects both neurotransmitter cycling and the broader energy status of the neuron, reinforcing a vicious cycle of excitotoxicity and metabolic stress

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