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The neurodegenerative effects of Meth result in part from inflammation initiated by the activation of astrocytes and microglia, increased production of COX-2, and consequent increases in proinflammatory prostaglandins and cytokines such as TNF-, IL1-, and IL-6 18

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Moreover, we explore the state of the art in the technological field of treating this fundamental symptom from current definitions to diagnosis and discuss procedures used in daily practice (clinical evaluation, EDSS, cognitive and neuropsychological tests)

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