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This tripeptide serves as the master regulator of cellular redox homeostasis and functions as a critical cofactor in numerous enzymatic reactions essential for life

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Treatment with a clinically-relevant dose of methylphenidate alters NMDA receptor composition and synaptic plasticity in the juvenile rat prefrontal cortex

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GSH-independent thioredoxin pathway), which may prevent GSH depletion-dependent oxidative stress and cell death 18